Case (
case) wrote in
fandomsecrets2013-01-17 06:47 pm
[ SECRET POST #2207 ]
⌈ Secret Post #2207 ⌋
Warning: Some secrets are NOT worksafe and may contain SPOILERS.
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Notes:
Secrets Left to Post: 01 pages, 023 secrets from Secret Submission Post #315.
Secrets Not Posted: [ 0 - broken links ], [ 0 - not!secrets ], [ 0 - not!fandom ], [ 0 - too big ], [ 0 - repeat ].
Current Secret Submissions Post: here.
Suggestions, comments, and concerns should go here.

Re: Things you eat that horrifies other
(Anonymous) 2013-01-18 06:31 am (UTC)(link)well,they do have all the canned foods that last forever so i'd figure 50/50? they're just so particular about what parts of the animal they eat O_O;;
Re: Things you eat that horrifies other
(Anonymous) 2013-01-18 07:13 am (UTC)(link)Re: Things you eat that horrifies other
(Anonymous) 2013-01-18 11:51 am (UTC)(link)Re: Things you eat that horrifies other
(Anonymous) 2013-01-18 03:25 pm (UTC)(link)Re: Things you eat that horrifies other
(Anonymous) 2013-01-18 03:31 pm (UTC)(link)That's how several people on both sides of my family have gotten it.
Re: Things you eat that horrifies other
(Anonymous) 2013-01-18 03:54 pm (UTC)(link)http://en.wikipedia.org/wiki/Gout
Dietary causes account for about 12% of gout,[2] and include a strong association with the consumption of alcohol, fructose-sweetened drinks, meat, and seafood.[4][8] Other triggers include physical trauma and surgery.[6] Recent studies have found dietary factors once believed associated are, in fact, not, including the intake of purine-rich vegetables (e.g., beans, peas, lentils, and spinach) and total protein.[9][10] The consumption of coffee, vitamin C and dairy products, as well as physical fitness, appear to decrease the risk.[11][12][13] This is believed partly due to their effect in reducing insulin resistance.[13]
http://en.wikipedia.org/wiki/Hyperuricemia
A purine-rich diet is a common but minor cause of hyperuricemia. Diet alone generally is not sufficient to cause hyperuricemia. Hyperuricemia of this type is a common complication of solid organ transplant.[6]
High intake of alcohol (ethanol), a significant cause of hyperuricemia, has a dual action that is compounded by multiple mechanisms. Ethanol increases production of uric acid by increasing production of lactic acid, hence lactic acidosis. Ethanol also increases the plasma concentrations of hypoxanthine and xanthine via the acceleration of adenine nucleotide degradation, and is a possible weak inhibitor of xanthine dehydrogenase. As a byproduct of its fermentation process, beer additionally contributes purines. Ethanol decreases excretion of uric acid by promoting dehydration and (rarely) clinical ketoacidosis.[3]
High dietary intake of fructose contributes significantly to hyperuricemia.[11][12][13] In a large study in the United States, consumption of four or more sugar-sweetened soft drinks per day gave an odds ratio of 1.82 for hyperuricemia.[14] Increased production of uric acid is the result of interference, by a product of fructose metabolism, in purine metabolism. This interference has a dual action, both increasing the conversion of ATP to inosine and hence uric acid and increasing the synthesis of purine.[15] Fructose also inhibits the excretion of uric acid, apparently by competing with uric acid for access to the transport protein SLC2A9.[16] The effect of fructose in reducing excretion of uric acid is increased in people with a hereditary (genetic) predisposition toward hyperuricemia and/or gout.[15]
Starvation causes the body to metabolize its own (purine-rich) tissues for energy. Thus, like a high purine diet, starvation increases the amount of purine converted to uric acid. A very low calorie diet without carbohydrate can induce extreme hyperuricemia; including some carbohydrate (and reducing the protein) reduces the level of hyperuricemia.[17] Starvation also impairs the ability of the kidney to excrete uric acid, due to competition for transport between uric acid and ketones.[18]